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Di­etary mag­ne­sium may help to lower el­e­vated gly­cated he­mo­glo­bin (HbA1c), el­e­vated sys­tolic blood pres­sure, and el­e­vated C-re­ac­tive pro­tein.. Mag­ne­sium is a min­eral with a ma­jor role in the me­tab­o­lism of glu­cose, the pro­duc­tion of cel­lu­lar energy, and the man­u­fac­ture of pro­tein. A re­search team led by Yanni Pa­paniko­laou (France), and col­leagues as­sessed data col­lected on sub­jects, ages 20 years and older, en­rolled in the US Na­tional Health and Nutri­tion Ex­am­i­na­tion Sur­vey (NHANES), 2001-2010. The team de­ter­mined mag­ne­sium in­take from foods alone, and from foods plus di­etary sup­ple­ments us­ing the meth­ods from the Na­tional Can­cer In­sti­tute. Adults with ad­e­quate in­take of mag­ne­sium from food had sig­nif­i­cantly dif­fer­ent HOMA-IR – a mea­sure of in­sulin re­sis­tance, sys­tolic blood pres­sure, and HDL-choles­terol, as com­pared to sub­jects with in­ad­e­quate in­take of mag­ne­sium from food. Ad­e­quate in­take of mag­ne­sium from food plus di­etary sup­ple­ment had sig­nif­i­cant dif­fer­ences in waist cir­cum­fer­ence, sys­tolic blood pres­sure, and HDL (high-den­sity lipopro­tein) choles­terol. The team ob­served that a higher di­etary in­take of mag­ne­sium from all sources as­so­ci­ated with “sig­nif­i­cantly re­duced odds ra­tios for el­e­vated gly­co­he­moglobin, meta­bolic syn­drome, obe­sity, over­weight or obe­sity, el­e­vated waist cir­cum­fer­ence, el­e­vated sys­tolic blood pres­sure, re­duced HDL and el­e­vated C-re­ac­tive pro­tein. The study au­thors sub­mit that: “there is a ben­e­fi­cial re­la­tion­ship be­tween di­etary mag­ne­sium in­take and di­a­betes-re­lated phys­i­o­log­i­cal out­comes.”

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